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A 70 something female with chest pain (KG- done)

Dr. Smith's ECG Blog

One study found that the best discrimination of stress cardiomyopathy from ACS was possible with the ratio of NT-proBNP/cTnT on the 2nd day. and an accuracy of ∼96% in detecting stress cardiomyopathy as opposed to ACS. NT-proBNP and CTnT in ACS and Takotsubo) Smith : However, this is not of any help with the acute diagnosis!

ACS 52
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Interns are not (yet) poisoned by the STEMI/NSTEMI paradigm

Dr. Smith's ECG Blog

A 41-year-old South Asian male with history of hypertension, alcohol use disorder and hyperlipidemia, who has a strong family history of CAD presented with central substernal burning, pressure, and pain with associated diaphoresis. Patient initially presented at 9 PM to a referring facility with hsTnI 13 (ref: < 34 ng/L) then 30, then 60.

STEMI 70
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ST Elevation in aVR

Dr. Smith's ECG Blog

They show that if there is not >/= 1 mm STE in aVR, then ACS is highly unlikely to be due to severe 3-Vessel disease or Left Main. Because if such severe CAD is present, the patient is likely to need CABG. Am J Cardiol;107(4):495-500. why is this important?

CAD 52
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Two Cases: Was it an error to activate the cath lab? Add AG case for 3rd one, except it is already listed as inferior aneurysm case.

Dr. Smith's ECG Blog

Multivessel CAD 2. Suspect type 2 NSTEMI, although difficult to rule out type 1 Given lack of ACS symptoms and multivessel coronary artery disease, her cause was paused for consideration of a heart team approach with consideration for CABG. Underlying right basilar atelectasis and or infiltrate cannot be excluded. cm (normal = 0.7

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Elder Male with Syncope

EMS 12-Lead

2. Coronary angiography reveals significant and severe CAD involving all three epicardial vessels. Sudden narrowing of a coronary artery due to ACS (plaque rupture with thrombosis and/or downstream showering of platelet-fibrin aggregates). He awoke earlier that morning in his usual state of health. He was taken to the Cath Lab.

Coronary 290
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An undergraduate who is an EKG tech sees something. The computer calls it completely normal. How about the physicians?

Dr. Smith's ECG Blog

A 63 year old man with a history of hypertension, hyperlipidemia, prediabetes, and a family history of CAD developed chest pain, shortness of breath, and diaphoresis after consuming a large meal at noon. Smith comment : Is the ACS (rupture plaque) with occlusion that is now reperfusing? Edited by Smith He also sent me this great case.

CAD 132
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Formula Utilization

EMS 12-Lead

Moreover, he had no pertinent medical history to report in terms of CAD, HTN, HLD, or DM, for example. Although the attending crews did not consider the ECG pathognomonic for occlusive thrombosis, they nonetheless considered the patient high-risk for ACS and implored him to reconsider. A 12 Lead ECG was recorded.

ACS 130